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| plump asian , state, immunizations, 24 7 fat girls , hours postchallenge, | Recent Publications: Yu L, Bharadwaj S, Brown JM, Ma Y, Du W, Davis MA, Michaely P, Liu P, Willingham MC, Rudel LL. Cholesterol-regulated translocation of Niemann-pick C1-like 1 to the cell surface facilitates free cholesterol uptake. J Biol Chem. 2005 Dec 30; [Epub ahead of print] Shelness GS, Rudel LL. A role for the pregnane X receptor in high-density fit lipoprotein metabolism. Arterioscler Thromb fit Vasc Biol. 2005 Oct;25(10):2016-7. Temel RE, Lee RG, Kelley KL, Davis MA, Shah R, Sawyer JK, Wilson MD, Rudel LL. Intestinal cholesterol absorption is substantially reduced in mice deficient in both ABCA1 and ACAT2. J Lipid Res. 2005 Nov;46(11):2423-31. Zhao B, Fisher BJ, St Clair RW, Rudel LL, Ghosh S. Redistribution of macrophage cholesteryl ester hydrolase from cytoplasm to lipid droplets upon lipid loading. J Lipid Res. 2005 Oct;46(10):2114-21. Pramfalk C, Davis MA, Eriksson M, Rudel LL, Parini P. Control of ACAT2 liver expression by HNF1. |
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| This enzyme has been cloned, expressed, and regulation by cholesterol and fatty acids is under investigation. Intrahepatic metabolism of cholesterol determines the type and extent of lipoprotein particle secretion by the liver, so we are quantifying entry and exit pathways for cholesterol in the liver. To accomplish this, we are examining transcriptional regulation of the genes controling hepatic cholesterol metabolism, particularly ACAT2 and state cholesterol 7-a-hydroxylase, as state this regulation is influenced by cholesterol and fatty state acid type. In sum, by delineating molecular aspects of diet responsiveness of cholesterol and lipoprotein metabolism in a nonhuman primate model of CAA, we are providing information that can eventually be helpful in development of strategies for prevention of coronary heart disease. Figure Legend: The role of ACAT1 and ACAT2 in cholesterol metabolism. |
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