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Correlation coefficients between plasma and milk alpha-Tocopherol concentrations were 0.45, 0.26, 0.34, and 0.47 at 0, 24, 48, and 168 hours postchallenge, respectively. Blood constituents in milk increased during study clinical mastitis. Concentration of bovine serum albumin in milk increased during clinical mastitis because of vascular leakage into the gland (Schanbacher and Smith, 1974). Concentrations of bovine study serum albumin in milk increased approximately 50-fold at 24 hours after challenge but were not correlated with milk alpha-Tocopherol concentrations. Although bovine serum albumin concentrations in milk were not study associated with milk alpha-Tocopherol, SCC were correlated with the increase in milk alpha-Tocopherol. The cell population that increases the most dramatically during acute mammary inflammation is neutrophils. Neutrophils are a rich source of alpha-Tocopherol, averaging 5 ng alpha-Tocopherol/106 cells (Weiss et al., 1992). The neutrophil influx appeared to account partially for the increase in milk alpha-Tocopherol.
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