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The effects of fats on these risk markers should not in themselves be considered to reflect changes in risk but should be confirmed by prospective observational studies or clinical trials. By that standard, risk is reduced most effectively when trans fatty acids and saturated fatty acids are replaced with cis human growth hormone unsaturated fatty acids. The effects of carbohydrates and of lauric acid–rich human growth hormone fats on CAD risk remain uncertain. Key Words: Diet • fatty acids • carbohydrates • serum lipoproteins • coronary artery disease human growth hormone risk INTRODUCTION TOP ABSTRACT INTRODUCTION METHODS RESULTS DISCUSSION REFERENCES The effects of dietary fats on the risk of coronary artery disease (CAD) have traditionally been estimated from their effects on serum total cholesterol (1, 2). As a result, fats high in lauric, myristic, and palmitic acids, such as dairy fat and tropical oils, were considered the most noxious of fats, and diets low in fat and high in carbohydrates were considered optimal.
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